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statement that haploinsufficiency of heparin cofactor II, a glycosaminoglycan-dependent thrombin inhibitor, exacerbates injury- or hyperlipidemia-induced arterial lesion formation in mice, possibly by excessive thrombin signaling through protease-activated receptors (see the related article beginning on page 1514)

statement that haploinsufficiency of heparin cofactor II, a glycosaminoglycan-dependent thrombin inhibitor, exacerbates injury- or hyperlipidemia-induced arterial lesion formation in mice,…

The utilization during protein interaction analysis of stringent filtering to be able to select a rank for the confidence of interactions (for instance potential and/or high-confidence interactions) and of additional precautions like the exclusion of baits showing unusually lot of background proteins (see [17]) is essential in these studies for the reason why reported above [44]

The utilization during protein interaction analysis of stringent filtering to be able to select a rank for the confidence of…

2004;201:109C137

2004;201:109C137. GUID:?EA6123EF-64F2-45AF-850A-06953924D885 Summary The molecular mechanisms controlling the termination of cortical interneuron migration are unknown. Here we demonstrate that prior…